Service interruption on Monday 11 July from 12:30 to 13:00: all the sites of the CCSD (HAL, EpiSciences, SciencesConf, AureHAL) will be inaccessible (network hardware connection).
Skip to Main content Skip to Navigation
Journal articles

TAFA4 relieves injury-induced mechanical hypersensitivity through LDL receptors and modulation of spinal A-type K+ current

Abstract : Highlights d TAFA4 reverses inflammatory, postoperative, and nerve injury-induced mechanical pain d TAFA4-mediated antihypersensitive effect requires functional LDL receptor family members d SNI alters spinal I A in opposite direction in excitatory and inhibitory interneurons d TAFA4 reverses SNI-induced alteration of I A in both interneuron subtypes
Document type :
Journal articles
Complete list of metadata

https://hal-amu.archives-ouvertes.fr/hal-03406182
Contributor : Sébastien Poulain Connect in order to contact the contributor
Submitted on : Wednesday, October 27, 2021 - 5:01:50 PM
Last modification on : Friday, May 20, 2022 - 3:41:13 AM
Long-term archiving on: : Friday, January 28, 2022 - 7:24:45 PM

File

TAFA4 relieves injury-induced ...
Publisher files allowed on an open archive

Licence


Distributed under a Creative Commons Attribution - NonCommercial - NoDerivatives 4.0 International License

Identifiers

Collections

Citation

Sungjae yoo, Catarina Santos, Ana Reynders, Irène Marics, Pascale Malapert, et al.. TAFA4 relieves injury-induced mechanical hypersensitivity through LDL receptors and modulation of spinal A-type K+ current. Cell Reports, Elsevier Inc, 2021, 37 (4), pp.109884. ⟨10.1016/j.celrep.2021.109884⟩. ⟨hal-03406182⟩

Share

Metrics

Record views

30

Files downloads

16